It may be a vascular disease.
That would explain a lot. We’ve been comparing it to the flu, but it’s not a good comparison.
9 thoughts on “The Virus”
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It may be a vascular disease.
That would explain a lot. We’ve been comparing it to the flu, but it’s not a good comparison.
Comments are closed.
It’s the Andromeda Strain.
If first kills by infecting your blood vessels linings so you clot.
It then mutates that it crashes recce aircraft by starving the pilot of oxygen by eating the silicone parts on the oxygen mask. It also eats the seals in the Wildfire lab, setting the atom bomb countdown that the youngest of the scientists has to crawl into the central core while getting shot with tranquilizer darts meant to stop monkeys.
We need to wait for the right storm conditions to wash it out to sea, where the alkaline pH wipes it out.
I see from the article those statins I’d rather not take may also help protect me from the effects of Chinese Bat Cooties.
I find this article pretty convincing. I’ve often wondered why Wuhan Virus patients often have low blood O2 levels but don’t feel it; vascular inflammation would probably explain that.
Paul, my first thought, exactly. But you beat me to it!
Almost, but unlike A-Strain, coronavirus has RNA.
A riveting book. A mediocre movie.
Faster please. My big worry is that COVID-19 syndrome will disappear before these treatments can be tested for effectiveness.
Essentially this is what happened with SARS. And now here we are (again).
People think viruses are essentially static. Nothing could be further from the truth. We will either have to go to parts of the world where there are still outbreaks to test, or induce the symptoms using live virus on volunteers. I sure hope we can avoid the latter.
I have said this before, but I think that in a virus outbreak, the virus being passed from one person to the next “attenuates” it enough that becomes kind of like a live virus vaccine. Only for a live virus vaccine like the oral polio vaccine, it is passed through multiple generates of a non-human animal host, first?
As I have also said, in Colonial days prior to the Jenner cowpox inoculation, wealthy people would “pass on” smallpox through their servants before inoculating their own children with it? Another version of this is “viral load”, that the inoculation with live smallpox was done with a small amount?
So if the virus “attenuates” by a viral “game of telephone” on being passed from one human host to the next, where does the next measles or mumps outbreak come from? Are there reservoirs of the base virus strain to start the cycle over again.
Yes, herd immunity once measles runs its course, and once that population ages and there is a new generation of the susceptible, a new epidemic breaks out. But it seems there is more to it than that.
“In SARS1, the protein that’s required to cleave it is likely present only in the lung environment, so that’s where it can replicate. To my knowledge, it doesn’t really go systemic,” Lee says. “[SARS-CoV-2] is cleaved by a protein called furin, and that’s a big danger because furin is present in all our cells, it’s ubiquitous.”
Gee, if I were working hard to “amplify” a virus, what area(s) would I focus on?
I understand the virus suppresses production of interferons by infected cells (it has an RNAse that destroys the cell’s messenger RNA, stopping synthesis of the interferons, but the virus’ own RNA has a “hairpin” that prevents the enzyme from touching it.)
So, this suggests that artificially supplementing a patient with interferons (1 and 3, I believe) would counter this suppression. For some reason this is being done more overseas than here.